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For 高評価なギフト Ford Expedition 1997 【格安SALEスタート】 Rear 1998 Driveshaft

For Ford Expedition 1997 1998 Rear Driveshaft

$90

For Ford Expedition 1997 1998 Rear Driveshaft

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Item specifics

Condition:
New
Brand:
BuyAutoParts
Manufacturer Part Number:
91-01760AN
Warranty:
1 Year
Interchange Part Number:
19101760-101, F75Z4602RA ; XL1Z4602BA
UPC:
193331011798

For Ford Expedition 1997 1998 Rear Driveshaft

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a leading international journal from BMJ and BSG, publishes cutting-edge gastroenterology and hepatology research and reviews

Impact Factor: 23.059
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Gut is a Plan S compliant Transformative Journal.

Gut is a leading international journal in gastroenterology and hepatology and has an established reputation for publishing first class clinical research of the alimentary tract, the liver, biliary tree and pancreas. Gut delivers up-to-date, authoritative, clinically oriented coverage in all areas of gastroenterology and hepatology. Regular features include articles by leading authorities describing novel mechanisms of disease and new management strategies, both diagnostic and therapeutic, likely to impact on clinical practice within the foreseeable future.

Gut is an official journal of the British Society of Gastroenterology and has two companion titles: Frontline Gastroenterology for education and practice and BMJ Open Gastroenterology for sound science clinical research.

Editor-in-Chief: Professor Emad El-Omar, University of New South Wales, Sydney, Australia
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Synergistic targeting and resistance to PARP inhibition in pancreatic cancer

To cite: Gout J, Perkhofer L, Morawe M, et al. Gut 2021;70:743-760.

Read the full article here: link
Objective: ATM serine/threonine kinase (ATM) is the most frequently mutated DNA damage response gene, involved in homologous recombination (HR), in pancreatic ductal adenocarcinoma (PDAC).

Conclusion: Analysis of the molecular susceptibilities triggered by ATM deficiency in PDAC allow elaboration of an efficient mutation-specific combinational therapeutic approach that can be also implemented in a genotype-independent manner by ATM inhibition.

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